Try drinking it under a tree?

Joe, you forgot to mention the natural IQ boosters in cannabis...I think its scientifically referred to as...stupidogenic and moronimetic effects!

Im still laughing....

yeah sure...

Cannabis compound can help brain cells

In a paper published in the Journal of Neuroscience, the researchers report on their studies into cannabidiol – a naturally occurring molecule found in cannabis.

Also known as CBD, it is not the constituent that gives the high – that compound is called tetrahydrocannabinol or THC – and so may be more acceptable as a drug treatment.

Both compounds are currently used in a pharmaceutical medicine to help patients relieve pain and other symptoms of Multiple Sclerosis.

Now researchers have discovered how CBD actually works within brain cells.

By interacting with mitochondria – which are the power generators of all cells – it can help maintain normal levels of calcium allowing cells to function properly and providing a greater resistance to damage.

Disturbance of calcium levels has long been associated with a number of brain disorders. So the finding could have implications for the development of new treatments for disorders related to malfunctioning mitochondria.

http://www.jneurosci.org/content/29/7/2053.full?sid=9f67a71f-c0a7-4bf0-b66c-6add8da76b7d

Pot Compound Protects Against Alcohol-Induced Brain Damage

Researchers hypothesized that CBD is neuroprotective because it possesses anti-oxidant properties. Anti-oxidants, such as vitamin C and vitamin E, are believed to help the body protect against the deleterious effects of free radicals (unstable atoms that can damage cells and may accelerate the progression of cancer and age-related diseases).

H t t p://jpet.aspetjournals.org/content/314/2/780.abstract?sid=4498b396-f35c-49c8-9709-900ffa17e299

Yeah, Joe, next you're going to tell us that it cures throat cancer because Michael Douglas says so. Look many of us have smoked weed in our lifetime. Trust me, you're not convincing anyone that weed is a cure all. What ever benefits you receive in one area are most definitely detracting from others. BTW, don't drive, operate heavy machinery or attempt to have a coherent and intelligent conversation while "medicated" with cannabis/weed!

uhh...it does cure cancer...and it's not dangerous to smoke it up and drive...sober drivers are just as dangerous as stoners...all you have to do is learn from the knowledge that is out there and make a better informed opinion instead of ranting with baseless claims...

Studies assessing the anticancer properties of cannabinoids have shown that they inhibit the proliferation of a wide range of cancers, including brain cancer, prostate cancer, oral cancers, lung cancer, skin cancer, pancreatic cancer, biliary tract cancers, lymphoma, and breast cancer.

The dual effects of delta(9)-tetrahydrocannabinol on cholangiocarcinoma (biliary tract cancer) cells: anti-invasion activity at low concentration and apoptosis induction at high concentration.

The anticancer effect of Delta (9)-tetrahydrocannabinol (THC), the principal active component of cannabinoids has been demonstrated in various kinds of cancers.

http://www.ncbi.nlm.nih.gov/pubmed/19916793

Cannabinoids inhibit cellular respiration of human oral cancer cells.

http://www.ncbi.nlm.nih.gov/pubmed/20516734

Anti-proliferative and apoptotic effects of anandamide in human prostatic cancer cell lines: implication of epidermal growth factor receptor down-regulation and ceramide production.

RESULTS: ANA induced a decrease of EGFR levels on LNCaP, DU145, and PC3 prostatic cancer cells by acting through cannabinoid CB(1) receptor subtype and this leaded to an inhibition of the EGF-stimulated growth of these cells.

http://www.ncbi.nlm.nih.gov/pubmed/12746841?dopt=Abstract

Cannabinoids reduce ErbB2-driven breast cancer progression through Akt inhibition

Results

Our results show that both Δ⁹-tetrahydrocannabinol, the most abundant and potent cannabinoid in marijuana, and JWH-133, a non-psychotropic CB₂ receptor-selective agonist, reduce tumor growth, tumor number, and the amount/severity of lung metastases in MMTV-neu mice. Histological analyses of the tumors revealed that cannabinoids inhibit cancer cell proliferation, induce cancer cell apoptosis, and impair tumor angiogenesis. Cannabinoid antitumoral action relies, at least partially, on the inhibition of the pro-tumorigenic Akt pathway. We also found that 91% of ErbB2-positive tumors express the non-psychotropic cannabinoid receptor CB₂.

http://www.molecular-cancer.com/content/9/1/196

Inhibition of skin tumor growth and angiogenesis in vivo by activation of cannabinoid receptors

http://www.jci.org/articles/view/16116/version/1

Δ⁹-Tetrahydrocannabinol inhibits epithelial growth factor-induced lung cancer cell migration in vitro as well as its growth and metastasis in vivo

http://www.nature.com/onc/journal/v27/n3/abs/1210641a.html

Cannabinoid Receptor-Mediated Apoptosis Induced by R(+)-Methanandamide and Win55,212-2 Is Associated with Ceramide Accumulation and p38 Activation in Mantle Cell Lymphoma

We have recently shown that cannabinoids induce growth inhibition and apoptosis in mantle cell lymphoma (MCL), a malignant B-cell lymphoma that expresses high levels of cannabinoid receptor types 1 and 2 (CB₁ and CB₂).

http://molpharm.aspetjournals.org/content/70/5/1612.abstract

Cannabinoids as potential new therapy for the treatment of gliomas (brain cancer)

http://www.expert-reviews.com/doi/abs/10.1586/14737175.8.1.37

Cannabinoids Induce Apoptosis of Pancreatic Tumor Cells via Endoplasmic Reticulum Stress–Related Genes

http://cancerres.aacrjournals.org/content/66/13/6748.abstract

Neurophysiological and cognitive effects of smoked marijuana in frequent users.
Overall performance accuracy was not significantly altered by marijuana
http://www.ncbi.nlm.nih.gov/pubmed/20600251?dopt=Abstract

STONERS AND DRIVING:

"[In] cases in which THC was the only drug present were analyzed, the culpability ratio was found to be not significantly different from the no-drug group."

REFERENCE: G. Chesher and M. Longo. 2002. Cannabis and alcohol in motor vehicle accidents. In: F. Grotenhermen and E. Russo (Eds.) Cannabis and Cannabinoids: Pharmacology, Toxicology, and Therapeutic Potential. New York: Haworth Press. Pp. 313-323.

"Cannabis leads to a more cautious style of driving, [but] it has a negative impact on decision time and trajectory. [However,] this in itself does not mean that drivers under the influence of cannabis represent a traffic safety risk. … Cannabis alone, particularly in low doses, has little effect on the skills involved in automobile driving."

REFERENCE: Canadian Senate Special Committee on Illegal Drugs. 2002. Cannabis: Summary Report: Our Position for a Canadian Public Policy. Ottawa. Chapter 8: Driving Under the Influence of Cannabis.

"This report has summarized available research on cannabis and driving.
… Evidence of impairment from the consumption of cannabis has been reported by studies using laboratory tests, driving simulators and on-road observation. ... Both simulation and road trials generally find that driving behavior shortly after consumption of larger doses of cannabis results in (i) a more cautious driving style; (ii) increased variability in lane position (and headway); and (iii) longer decision times. Whereas these results indicate a 'change' from normal conditions, they do not necessarily reflect 'impairment' in terms of performance effectiveness since few studies report increased accident risk."

REFERENCE: UK Department of Environment, Transport and the Regions (Road Safety Division). 2000. Cannabis and Driving: A Review of the Literature and Commentary. Crowthorne, Berks: TRL Limited.

"Overall, we conclude that the weight of the evidence indicates that:
1) There is no evidence that consumption of cannabis alone increases the risk of culpability for traffic crash fatalities or injuries for which hospitalization occurs, and may reduce those risks.
2) The evidence concerning the combined effect of cannabis and alcohol on the risk of traffic fatalities and injuries, relative to the risk of alcohol alone, is unclear.
3) It is not possible to exclude the possibility that the use of cannabis (with or without alcohol) leads to an increased risk of road traffic crashes causing less serious injuries and vehicle damage."

REFERENCE: M. Bates and T. Blakely. 1999. "Role of cannabis in motor vehicle crashes." Epidemiologic Reviews 21: 222-232.

"In conclusion, marijuana impairs driving behavior. However, this impairment is mitigated in that subjects under marijuana treatment appear to perceive that they are indeed impaired. Where they can compensate, they do, for example by not overtaking, by slowing down and by focusing their attention when they know a response will be required. … Effects on driving behavior are present up to an hour after smoking but do not continue for extended periods.
With respect to comparisons between alcohol and marijuana effects, these substances tend to differ in their effects. In contrast to the compensatory behavior exhibited by subjects under marijuana treatment, subjects who have received alcohol tend to drive in a more risky manner. Both substances impair performance; however, the more cautious behavior of subjects who have received marijuana decreases the impact of the drug on performance, whereas the opposite holds true for alcohol."

REFERENCE: A. Smiley. 1999. Marijuana: On-Road and Driving-Simulator Studies. In: H. Kalant et al. (Eds) The Health Effects of Cannabis. Toronto: Center for Addiction and Mental Health. Pp. 173-191.

CRASH CULPABILITY STUDIES

“For each of 2,500 injured drivers presenting to a hospital, a blood sample was collected for later analysis.

There was a clear relationship between alcohol and culpability. … In contrast, there was no significant increase in culpability for cannabinoids alone. While a relatively large number of injured drivers tested positive for cannabinoids, culpability rates were no higher than those for the drug free group. This is consistent with other findings.”

REFERENCE: Logan, M.C., Hunter, C.E., Lokan, R.J., White, J.M., & White, M.A. (2000). The Prevalence of Alcohol, Cannabinoids, Benzodiazepines and Stimulants Amongst Injured Drivers and Their Role in Driver Culpability: Part II: The Relationship Between Drug Prevalence and Drug Concentration, and Driver Culpability. Accident Analysis and Prevention, 32, 623-32.

“Blood samples from 894 patients presenting to two Emergency Departments for treatment of motor vehicle injur[ies] … were tested for alcohol and other drugs.

… Based on alcohol and drug testing of the full range of patients … alcohol is clearly the major drug associated with serious crashes and greater injury. Patients testing positive for illicit drugs (marijuana, opiates, and cocaine), in the absence of alcohol, were in crashes very similar to those of patients with neither alcohol nor drugs. When other relevant variables were considered, these drugs were not associated with more severe crashes or greater injury.”

REFERENCE: P. Waller et al. 1997. Crash characteristics and injuries of victims impaired by alcohol versus illicit drugs. Accident Analysis and Prevention 29: 817-827.

“Blood specimens were collected from a sample of 1,882 drivers from 7 states, during 14 months in the years 1990 and 1991. The sample comprised operators of passenger cars, trucks, and motorcycles who died within 4 hours of their crash.

… While cannabinoids were detected in 7 percent of the drivers, the psychoactive agent THC was found in only 4 percent. … The THC-only drivers had a responsibility rate below that of the drugfree drivers. … While the difference was not statistically significant, there was no indication that cannabis by itself was a cause of fatal crashes.”

REFERENCE: K. Terhune. 1992. The incidence and role of drugs in fatally injured drivers. Washington, DC: US Department of Transportation National Highway Traffic Safety Administration, Report No. DOT HS 808 065.

ON-ROAD PERFORMANCE STUDIES

“Marijuana's effects on actual driving performance were assessed in a series of three studies wherein dose-effect relationships were measured in actual driving situations that progressively approached reality.

… THC's effects on road-tracking after doses up to 300 µg/kg never exceeded alcohol's at bacs of 0.08%; and, were in no way unusual compared to many medicinal drugs. Yet, THC's effects differ qualitatively from many other drugs, especially alcohol. Evidence from the present and previous studies strongly suggests that alcohol encourages risky driving whereas THC encourages greater caution, at least in experiments. Another way THC seems to differ qualitatively from many other drugs is that the formers users seem better able to compensate for its adverse effects while driving under the influence.”

REFERENCE: H. Robbe. 1995. Marijuana’s effects on actual driving performance. In: C. Kloeden and A. McLean (Eds) Alcohol, Drugs and Traffic Safety T-95. Adelaide: Australia: HHMRC Road Research Unit, University of Adelaide. Pp. 11-20.

“This report concerns the effects of marijuana smoking on actual driving performance. … This program of research has shown that marijuana, when taken alone, produces a moderate degree of driving impairment which is related to consumed THC dose. The impairment manifests itself mainly in the ability to maintain a lateral position on the road, but its magnitude is not exceptional in comparison with changes produced by many medicinal drugs and alcohol. Drivers under the influence of marijuana retain insight in their performance and will compensate when they can, for example, by slowing down or increasing effort. As a consequence, THC’s adverse effects on driving performance appear relatively small.”

REFERENCE: W. Hindrik and J. Robbe and J. O’Hanlon. 1993. Marijuana and actual driving performance. Washington, DC: US Department of Transportation National Highway Traffic Safety Administration, Report No. DOT HS 808 078.

TABULATED SUMMARY OF ROAD TRIALS OF CANNABIS AND DRIVING
Table compiled by the UK Department of Transport (2000)

DRIVING SIMULATOR STUDIES

“Overall, it is possible to conclude that cannabis has a measurable effect on psychomotor performance, particularly tracking ability. Its effect on higher cognitive functions, for example divided attention tasks associated with driving, appear not to be as critical. Drivers under the influence of cannabis seem aware that they are impaired, and attempt to compensate for this impairment by reducing the difficulty of the driving task, for example by driving more slowly.

In terms of road safety, it cannot be concluded that driving under the influence of cannabis is not a hazard, as the effects of various aspects of driver performance are unpredictable. However, in comparison with alcohol, the severe effects of alcohol on the higher cognitive processes of driving are likely to make this more of a hazard, particularly at higher blood alcohol levels.”

REFERENCE: B. Sexton et al. 2000. The influence of cannabis on driving: A report prepared for the UK Department of the Environment, Transport and the Regions (Road Safety Division). Crowthorne, Berks: TRL Limited.

btw, i'm high as hell right now...how's that for "coherent" or "intelligent" conversation? rebuttal? retort? breathlessly waiting...

uhh...it does cure cancer...and it's not dangerous to smoke it up and drive...sober drivers are just as dangerous as stoners...all you have to do is learn from the knowledge that is out there and make a better informed opinion instead of ranting with baseless claims...

Driving stoned on cannabis is equally dangerous as driving sober....Hold on while I fall out of my chair laughing at your ASININE BACK ASS-WARD HICK FROM THE STICKS LOGIC!!!!!!!!!!

Here is all the research you need, BURNOUT! I conducted my own extensive research years ago which is in line with EJELONU's!!! Now, go play in traffic, LMFAO!!!

HOW DOES MARIJUANA AFFECT THE BRAIN?

AKUDO EJELONU

Pot, weed, grass, ganja and skunk, are some of the common words used to describe the dried leaves drug known as marijuana. Marijuana is a cannabis plant that is "usually smoked or eaten to entice euphoria." (1). Throughout the years, there has been research on the negative and positive effects of marijuana on the human body and the brain. Marijuana is frequently beneficial to the treatment of AIDS, cancer, glaucoma, multiple sclerosis, and chronic pain. However, researchers such as Jacques-Joseph Moreau have been working to explain how marijuana has harmful affects on the functions of central nervous system and hinders the memory and movement of the user's brain. The focus of my web paper is how the chemicals in marijuana, specifically cannabinoids and THC have an effect on the memory and emotions of a person's central nervous system.

Marijuana impinges on the central nervous system by attaching to brain's neurons and interfering with normal communication between the neurons. These nerves respond by altering their initial behavior. For example, if a nerve is suppose to assist one in retrieving short-term memory, cannabinoids receptors make them do the opposite. So if one has to remember what he did five minutes ago, after smoking a high dose of marijuana, he has trouble. Marijuana plant contains 400 chemicals and 60 of them are cannabinoids, which are psychoactive compounds that are produced inside the body after cannabis is metabolized or is extorted from the cannabis plant. Cannabinoids is an active ingredient of marijuana. The most psychoactive cannabinoids chemical in marijuana that has the biggest impact on the brain is tetrahydrocannibol, or THC. THC is the main active ingredient in marijuana because it affects the brain by binding to and activating specific receptors, known as cannabinoid receptors. "These receptors control memory, thought, concentration, time and depth, and coordinated movement. THC also affects the production, release or re-uptake (a regulating mechanism) of various neurotransmitters."(2) Neurotransmitters are chemical messenger molecules that carry signals between neurons. Some of these affects are personality disturbances, depression and chronic anxiety. Psychiatrists who treat schizophrenic patient advice them to not use this drug because marijuana can trigger severe mental disturbances and cause a relapse.

When one's memory is affected by high dose of marijuana, short-term memory is the first to be triggered. Marijuana's damage to short-term memory occurs because THC alters the way in which information is processed by the hippocampus, a brain area responsible for memory formation. "One region of the brain that contains a lot of THC receptors is the hippocampus, which processes memory."(3) Hippocampus is the part of the brain that is important for memory, learning, and the integration of sensory experiences with emotions and motivation. It also converts information into short-term memory. "Because it is a steroid, THC acts on the hippocampus and inhibits memory retrieval."(4) THC also alters the way in which sensory information is interpreted. "When THC attaches to receptors in the hippocampus, it weakness the short-term memory,"(5) and damages the nerve cells by creating structural changes to the hippocampus region of the brain. When a user has a high dose of marijuana, new information does not register into their brain and this may be lost from memory and they are not able to retrieve new information for more than a few minutes. There is also a decrease in the activity of nerve cells.

There are two types of memory behavior that is affected by marijuana, recognition memory and free cells. Recognition memory is the ability to recognize correct words. Users can usually recognize words that they previous saw before smoking but claim to recognize words that they did not previously see before smoking. This mistake is known as memory intrusions. Memory intrusions are also the consequence of THC affecting the free cell of the brain. "Marijuana disrupts the ability to freely recall words from a list that has been presented to an intoxicated subject."(6) For example, if a list of vocabulary words presented to the intoxicated subject and few minutes later, they have to recall the words that were on the list. The only words that they remember are the last group of words and not the words that are in the beginning of the list. This is an initiation that their memory storage has been affected. "The absence of an effect at short term delay times indicates that cannabinodis did not impair the ability to perform the basic task, but instead produce a selective learning and/or memory deficit."(7) I did a study with two college students (Student A and Student B) who both smoke marijuana every other week. This particular study was done an hour before, while and after they were under the influence of the drug. Student A was watching television before she smoked marijuana, was asked which advertisement was splayed before the show started and she got four out of five of her answers correct. After this first section, she smoked a small dose of marijuana twice within an hour. Fifteen minutes after she smoked her last blunt, she continued her regular activity of watching sitcoms. When a commercial would come on, I would ask her simple questions like what happened before the show went to a commercial break. Her responses would be macro-answers about what was going on but when I asked her what the main character was wearing, she did not remember. This was ironic because the protagonist wore a bright yellow suit that my friend commenting on earlier when the show began ten minutes ago. Her short-term memory is weakening because she was only able to remember big picture information and not small picture. Though the results are interesting, I know that I would have had different response on someone else because it depends on how often the user smokes and if they have good memory prior to smoking weed.

Marijuana also impairs emotions. When smoking marijuana, the user may have uncontrollable laughter one minute and paranoia the next. This instant change in emotions has to do with the way that THC affects the brain's limbic system. The limbic system is another region of the brain that governs one's behavior and emotions. It also "coordinates activities between the visceral base-brain and the rest of the nervous system."(8) I am now going to use Students B to describe how emotions are affected by marijuana. Students B is an articulate and well spoken young woman who has a troublesome relationship with her best friends which gets her upset and tense up. But after she smoked one high dose weed, her body was relaxed however, she had trouble formulating her thoughts clearly and would talk in pieces and was jubilant. It has been researched that a person needs to have high dose of marijuana would be in the state of euphoria. High dose of marijuana is measured as "15mg of THC can cause increased heart rate, gross motor disturbances, and can lead to panic attacks."(9) Thankfully, Student A did not experience any of these extreme examples.

College students usually smoke marijuana because they are stressed over schoolwork and feel that marijuana can help them unwind. I have encountered marijuana smokers who are chilled and have no worries in the world but after the effect of the drug wears off, they're sometimes capable to tacking their problem or at the original state that they were in before the drug. The effects of happiness that marijuana usually cause to the user is not a lasting effect because even though a user smokes weed to get away from the troubles of his/her own life, they still have to face these problems after the effects of the drug wears-off. In a survey with college student, an organization called, parents: the Anti-Drug interviewed college students and found that "compared to the light users, heavy marijuana users made more errors and had more difficulty sustaining attention."(10) This was evident through my second experiment with Student B but not everyone who smoke high doses of marijuana experience the same affect.

The chemicals in marijuana bring cognitive impairment and troubles with learning for the user. "Smoking [marijuana] causes some changes in the brain that are like those caused but cocaine, heroin, and alcohol. Some researchers believe that has changes may put a person more at risk of becoming addicted to other drugs such a cocaine and heroin."(11) To prevent such harm, one must be cautious of their actions. Those who do not do drugs do not risk harm. So please the next day you light up, remember you that you central nervous system and brain will be at risk.

1)Online Dictionary

2)Marijuana: The Brain's Response to Drugs, A Good Web Source

3)Mind Over Matter: Marijuana Series, A Good Web Source

4)Alcohol Addiction & The Limbic System, A Good Web Source

5)Marijuana: The Brain's Response to Drugs, A Good Web Source

6)Cellular and Molecular Mechanisms Underlying Learning and Memory Impairments Produced by Cannabinoids, A Good Web Source

7)Cellular and Molecular Mechanisms Underlying Learning and Memory Impairments Produced by Cannabinoids, A Good Web Source

8)Marijuana and the Brain by John Gettman. High Times, March, 1995, A Good Web Source

9)Alcohol Addiction & The Limbic System, A Good Web Source

10)Parents. The Anti-Drug. -- Drug Information, A Good Web Source

11)Marijuana: Marijuana Brain Effects, A Good Web Source

Now I'm laughing kpokeefe. This thing reads like a middle school science report.

Please...none of the sources you cite are available anymore...they're all outdated and the only ones that actually opened were either outdated (citing works from 1985-1995) and does not address CBD's in marijuana, since it is a relatively recent discovery that offsets adverse effects of THC. in fact, the government holds a patent on CBD's (cannabidiols), and has had it since 2003...also, the other one opens to a page for parents regarding teenage use...this has nothing to do with responsible taxpaying adult recreational users...

The effects of herbal cannabis are a composite of a number of cannabinoid compounds, terpenoids and flavonoids. Thus, cannabidiol, a constituent of herbal cannabis, may offset some δ-9-THC effects (Zuardi et al., 1995). The ratio of the constituents of herbal cannabis varies, and this may result in important differences in its net effect.
http://www.namicalifornia.org/document-detail.aspx?page=newsviews&tabb=previousnews&lang=ENG&idno=126

Cannabis, of course, is more than THC. Other ingredients provide additional benefits and mitigate the adverse effects of THC.

Cannabidiol, for example, reduces dysphoria and depersonalization provoked by THC while contributing its own anxiolytic, antipsychotic, analgesic, antiemetic, anticarcinogenic, antioxidant, and neuroprotective effects.

http://www.jaoa.org/cgi/content/full/108/10/586

also, regarding memory loss and capacity...that only happens on a short-term basis. only to someone new to smoking marijuana experiences these effects...frequent users do not experience any loss of memory or mental capacity or coordination...your sources are outdated, let me update it for you:

Cannabinoid receptors are downregulated and desensitized when exposed to high doses of THC. This effect results in "drug tolerance," which occurs at varying rates and magnitudes in different brain regions. For example, it occurs faster and greater in the hippocampus, which regulates memory, compared with the basal ganglia, which mediates euphoric effect.²⁴ This difference may explain why memory loss decreases among frequent cannabis users, but its euphoric effects remain.²⁵

http://www.jaoa.org/cgi/content/full/108/10/586

7)Cellular and Molecular Mechanisms Underlying Learning and Memory Impairments Produced by Cannabinoids, A Good Web Source

this one is the one that's outdated...

10)Parents. The Anti-Drug. -- Drug Information, A Good Web Source

really? teenage studies?

That's right! A middle schooler can smoke a joint and know that he or she is HIGH. It doesn't taked a God D@#$ Phd to know that the properties of THC are mind altering! Do you know @!$%# stinks or are you one of those unfortunate people that has to have it hanging off the end of your nose before discovering?

Notice I said, "This is ALL the reasearch you'll need," EINSTEIN! Pot gives you a buzz just as Alcohol does as well as slowing the body's reflexes and reaction. Both lead to deaths operating a car while under the influence.

Finally, there isn't one shred of evidence that Pot cures cancer. It helps one stricken with it deal with the pain, PERIOD!

If it cured cancer, the government would be growing fields of it and selling a dime bag for $1000 to pay down the national debt...like tomorrow!!!

sure, just ignore the ones with the ph.d's...typical. raised by DARE.

also, the government IS growing bud and giving it to medical marijuana patients...they also approved of the THC prescription pill marinol and are approving a new prescription drug that blends THC and CBD's called sativex. so! the government recognizes the medical aspect! well well.

Joe, you're so HIGH you can't see the sarcasm in my post. You make my point for me as to the effects of THC and why driving while HIGH is a danger to everyone!!!

Get a life outside bubbling a bong!!!

also, the government IS growing bud and giving it to medical marijuana patients...they also approved of the THC prescription pill marinol and are approving a new prescription drug that blends THC and CBD's called sativex. so! the government recognizes the medical aspect! well well.

Joe, you're a LIAR! No private or government funded research has said that POT cures cancer! Show me on the FDA or CDC website explaining such research.

Link it up before you light it up, DUDE!

Phd's aren't hard to get! If you can support your claims in your dissertation as VALID then you get a piece of paper with your name followed by Phd. VALID doesn't mean its TRUE!

Critical thinking 101!

ok, so you want an offical government stance on the subject? here's one from NIDA...:

Q. What does marijuana do to the brain?

A. We know a lot about where marijuana acts in the brain and how it exerts its effects at specific sites called cannabinoid receptors. These are found in brain regions that influence learning and memory, appetite, coordination, and pleasure. That's why marijuana produces the effects it does. We know much less about what happens to the brain in the long run, when someone is a regular marijuana smoker.

Scientists use brain imaging techniques to study the living human brain, but we are still in the early stages of that research when it comes to marijuana...

Q. Isn't marijuana sometimes used as a medicine?

A.

...we know that the marijuana plant contains ingredients that could have important medical uses. Currently, two pill versions of THC, marijuana's psychoactive ingredient, have been approved to treat nausea in cancer chemotherapy patients and to stimulate appetite in some patients with AIDS. Also, a new product called Sativex is available in the United Kingdom and Canada as a mouth spray. It is a chemically pure mixture of plant-derived THC and cannabidiol (another chemical found in the marijuana plant)...

http://www.drugabuse.gov/marijbroch/teens/002b.php#q13

and the government is prepared to license growers to sell cannabis like tobacco:

Production, Analysis, & Distribution of Cannabis & Marijuana Cigarettes
Solicitation Number: N01DA-10-7773
Agency: Department of Health and Human Services
Office: National Institutes of Health
Location: National Institute on Drug Abuse

Synopsis:
Added: Aug 05, 2009 9:03 am
The National Institute on Drug Abuse is soliciting proposals from qualified organizations having the capability to (1) grow, harvest, analyze, store and distribute GMP grade cannabis (marijuana) on large and small scales; (2) extract cannabis to obtain purified phytocannabinoids including delta-9-tetrahydrocannabinol (delta-9-THC), analyze, and store; (3) prepare marijuana cigarettes and related products; and (4) distribute marijuana, marijuana cigarettes and cannabinoids, and other related products for research and other Government programs upon NIDA authorization.

https://www.fbo.gov/index?s=opportunity&mode=form&id=13b43512c37e45befa6e8f9556d276b0&tab=core&_cview=1

also, regarding the government growing and supplying medical marijuana:

Psst... Government-Supplied Marijuana Program Turns 30

That's right, our government has been supplying medical marijuana to some patients for three full decades.
May 7, 2008 |

Each month Irvin Rosenfeld goes to his pharmacy and picks up a special prescription, supplied to him by the U.S. government: a canister containing roughly 10 ounces of marijuana in pre-rolled cigarettes.

Rosenfeld, a Boca Raton, Florida stockbroker, suffers from a rare illness called multiple congenital cartilaginous exostosis, a painful genetic disease that causes tumors to grow at the ends of his long bones, causing unbelievable pain. He is also one of four surviving patients receiving government-supplied medical marijuana, in a program that was closed to new applicants by President George H.W. Bush in 1992.

That program marks its 30th anniversary May 10. That's right, our government has been supplying medical marijuana to a small number of patients -- the program peaked at 34 approved participants in 1991 -- for three full decades.

http://www.alternet.org/drugs/84766/?page=1

Irv Rosenfeld...He is one of the only four remaining patients whose cannabis is still provided by the federal government under its now disbanded Investigational New Drug (IND) program.

http://www.markbraunstein.org/articles/braunstein-cannabis-conference.pdf

The Compassionate Investigational New Drug program, or Compassionate IND, is a United States Federal Government-ran Investigational New Drug program that allows a limited number of patients to use medical marijuana grown at the University of Mississippi. It is administered by the National Institute on Drug Abuse. Closed to new entrants, there are only four surviving patients who were grandfathered into the program.^[1][2]

http://en.wikipedia.org/wiki/Compassionate_Investigational_New_Drug_program

still smokin it up here, "dude"...

Critical thinking 101!

lol...i think you'd have trouble passing that course...

also, patent #6630507...look it up sometime...the government has held the patent since 2003 on CBD's, a property of marijuana...so they know the medical benefits of marijuana...

Cannabinoids as antioxidants and neuroprotectants

Abstract

Cannabinoids have been found to have antioxidant properties, unrelated to NMDA receptor antagonism. This new found property makes cannabinoids useful in the treatment and prophylaxis of wide variety of oxidation associated diseases, such as ischemic, age-related, inflammatory and autoimmune diseases. The cannabinoids are found to have particular application as neuroprotectants, for example in limiting neurological damage following ischemic insults, such as stroke and trauma, or in the treatment of neurodegenerative diseases, such as Alzheimer's disease, Parkinson's disease and HIV dementia. Nonpsychoactive cannabinoids, such as cannabidoil, are particularly advantageous to use because they avoid toxicity that is encountered with psychoactive cannabinoids at high doses useful in the method of the present invention. A particular disclosed class of cannabinoids useful as neuroprotective antioxidants is formula (I) wherein the R group is independently selected from the group consisting of H, CH.sub.3, and COCH.sub.3. ##STR1##

Inventors:
Hampson; Aidan J. (Irvine, CA), Axelrod; Julius (Rockville, MD), Grimaldi; Maurizio (Bethesda, MD)

Assignee:
The United States of America as represented by the Department of Health and Human Services (Washington, DC)

Appl. No.:
09/674,028

Filed:
February 2, 2001

PCT Filed:
April 21, 1999

PCT No.:
PCT/US99/08769

PCT Pub. No.:
WO99/53917

PCT Pub. Date:
October 28, 1999

Joe, you're so HIGH you can't see the sarcasm in my post. You make my point for me as to the effects of THC and why driving while HIGH is a danger to everyone!!!

refer to post #3.5 ....

Pot gives you a buzz just as Alcohol does as well as slowing the body's reflexes and reaction. Both lead to deaths operating a car while under the influence.

nope. not true at all. again, refer to #3.5 and see that it is just as dangerous to drive sober as it is to drive stoned. that is not dangerous at all. in fact, coordination is not affected...

Neurophysiological and cognitive effects of smoked marijuana in frequent users.
Overall performance accuracy was not significantly altered by marijuana
http://www.ncbi.nlm.nih.gov/pubmed/20600251?dopt=Abstract

whoops, forgot the link for the CBD patent...

http://patft.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=6630507.PN.&OS=PN/6630507&RS=PN/6630507

no problem. smoke a bowl...we got you covered.

Wait... now we have to put warning labels on news articles to protect you from...... yourself? You truly think it's necessary to warn alcoholics that alcohol can be addictive? Wow...... I think I need a drink to follow that logic...

I somewhat agree. Usually, whatever health benefits are obtained by drinking wine can also be obtained by drinking grape juice, but is never explained in the article. Most of the studies are probably government funded and encouraging people to buy more wine will put more money in the treasury via the sin taxes placed on alcohol.

I'm pretty sure the article started off with the something along the lines of "you could also use a good dose of SPF" - so it doesn't make alcohol the only option. Good common sense should tell an alcoholic that the SPF option is the better way to go.

mohican - as one who has worked on government funded studies, I can tell you that the end game is to learn more about the brain - not to put more money in the treasury. Increasing government profits has never been a factor in the research. Otherwise that's an ethical issue, and the authors wouldn't be able to publish without disclosing third party interests. Believe it or not, not everything is a major conspiracy.

Dude...you're repeating yourself. Joe, I think he took your advice.

They're not on the highway, they're somewhere out in the sun. You really need to relax more. Is it lonely on that pedestal?

I find it interesting the article chose to highlight "Drink more wine" instead of "Eat more grapes"

I do think encouraging people to drink is not needed. Encouraging healthy eating IS.

So....to answer your question "one and only" -the answer is: NO. AKRANDY is not lonely on the pedestal.

But Toasty - I'm sure your skin was fine without the wine........

Why on Earth are YOU bothering to blog.

You sound to smart for this S#!+. :)

Halloween is a good time to read the bible. Judges 11:29-40 - human sacrifice of a virgin girl to the Judeo-Christian-Muslim god. Genesis 34:25 - mass murder. Numbers 15:32-36 - god orders death to a man picking up sticks!

Ecclesiastes 9:7 - "Go thy way, eat thy bread with joy, and drink thy wine with a merry heart; for God now accepteth thy works."